2017: The Effectiveness of tDCS/tACS/tRNS and pEMF Stimulation on EEG Neurotherapy Performance (Plenary Session)

In this session a discussion regarding neurostimulation technology will be conducted. The speaker will show how neurostimulation that is synchronized with EEG neurotherapy can enhance and facilitate regulation of the brain based on QEEG assessment and behavioral observational data. The limitations of EEG neurofeedback prevent adequate treatment outcomes on “tough cases” (such as autism) and decrease the credibility of the field in general when no treatment effects are observed. One of the main reasons for poor neurofeedback outcomes is associated with deregulation of the human connectome, or rich club. The human connectome has been strongly associated with the default mode network, and deregulation of this network can lead to poor communication between important network hubs that are responsible for regulating the brain. The speaker will discuss the current MRI BOLD research associated with physiological responses to pEMF, tDCS/tACS/tRNS stimulation. In 2016 multiple studies were published that show how stimulation technology triggers calcium ion activation in the glia which gives rise to global neuroplasticity responses. This new data supports the use of neurostimulation methods as they can help to facilitate changes in absolute power, coherence and phase towards the norm. The speaker will show and discuss QEEG and outcome data associated with clinical cases diagnosed with Autism, ADHD, Depression, Anxiety and Parkinson’s disease. Each of the cases discussed with include a summary of patient information, diagnosis, history and treatment plan. Each case will be presented with pre and post treatment EEG data. Behavioral changes will be discussed showing the effectiveness of EEG neurofeedback.

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In this session a discussion regarding neurostimulation technology will be conducted. The speaker will show how neurostimulation that is synchronized with EEG neurotherapy can enhance and facilitate regulation of the brain based on QEEG assessment and behavioral observational data. The limitations of EEG neurofeedback prevent adequate treatment outcomes on “tough cases” (such as autism) and decrease the credibility of the field in general when no treatment effects are observed. One of the main reasons for poor neurofeedback outcomes is associated with deregulation of the human connectome, or rich club. The human connectome has been strongly associated with the default mode network, and deregulation of this network can lead to poor communication between important network hubs that are responsible for regulating the brain. The speaker will discuss the current MRI BOLD research associated with physiological responses to pEMF, tDCS/tACS/tRNS stimulation. In 2016 multiple studies were published that show how stimulation technology triggers calcium ion activation in the glia which gives rise to global neuroplasticity responses. This new data supports the use of neurostimulation methods as they can help to facilitate changes in absolute power, coherence and phase towards the norm. The speaker will show and discuss QEEG and outcome data associated with clinical cases diagnosed with Autism, ADHD, Depression, Anxiety and Parkinson’s disease. Each of the cases discussed with include a summary of patient information, diagnosis, history and treatment plan. Each case will be presented with pre and post treatment EEG data. Behavioral changes will be discussed showing the effectiveness of EEG neurofeedback.

2017: The Effectiveness of tDCS/tACS/tRNS and pEMF Stimulation on EEG Neurotherapy Performance (Plenary Session)
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